Train Like A Pro! :)
February 14
- Health data: Sick: 3
February 13
February 12
Trainer ride
20min warm up at 150
65 minutes as 5min/85%/5min 75%
Cool down
Total average power: 174 watts. Great solid ride after yesterday's effort!
Strong run in some really cold, windy conditions. I was stupidly under-dressed- damn windchill! Makes you run faster, though!
Did this run 3 hours post-bike, when I finally had enough food in me to exercise again. I ate 2 gu's on the bike, that's how hungry I've been! I had 5 gu's and 1 bag of chomps on the bike yesterday- jeesh!
February 11
Trainer ride
Warm up 20min @154 watts
1:10 at 178 watts
3 min break at 164
1:00 at 180 watts
2 min break
20 minutes at 202 watts. That nearly killed me. :)
5 min cool down at 147 watts.
Total average power: 176 watts.
Fun run with Cody and Ryan in the dark, snowy night. Good times!
Run stability and core
February 10
Run test. 2.43 miles warm up to the track (city streets- yuck!)
800 all out- 2:54 (5:44min/mile)
1 easy lap
2 miles all out- 12:19. Interestingly, my garmin had my pace at 6:03. I ran this at the track, though, and started and stopped in the same place. On the track, this was 2 miles, but according to garmin, it was 2.04. That's a big difference in pace!
Main set- 10x200. Whoa that was hard. Tried to get every 200 under 3:00, and some were even at 2:50. The pool was super crowded, so it wasn't exactly easy to have a consistent set. My arms felt like they were going to fall off at the end, but this was a great swim!
February 9
Failed. Today was a workout failure. Oh well!
And here is the abstract, for your reading pleasure.....heh
Beta oscillations in cortical-basal ganglia (BG) circuits have been implicated in normal movement suppression and motor impairment in Parkinson’s disease. To dissect the functional correlates of these rhythms we compared neural activity during four distinct variants of a cued choice task in rats. Brief beta (20 Hz) oscillations occurred simultaneously throughout the cortical-BG network, both spontaneously and at precise moments of task performance. Beta phase was rapidly reset in response to salient cues, yet increases in beta power were not rigidly linked to cues, movements, or movement suppression. Rather, beta power was enhanced after cues were used to determine motor output. We suggest that beta oscillations reflect a postdecision stabilized state of cortical-BG networks, which normally reduces interference from alternative potential actions. The abnormally strong beta seen in Parkinson’s Disease may reflect overstabilization of these networks, producing pathological persistence of the current motor state.
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